What will kill the hiv virus
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Britney Spears Responded To Sister. Hood, a research instructor in medicine at WU, said in a press release. The virus has to have a protective coat, a double-layered membrane that covers the virus. Because HIV cells are smaller than regular cells, they slide between the bumpers while leaving healthy, normal cells intact.
Researchers say these bee venom nanoparticles could be used in a vaginal gel to help prevent the spread of HIV in developing countries, such as parts of Africa with a high HIV rate. They could also be used by people who want HIV protection, but not contraception. Beyond preventive measures, Hood sees the potential for treating existing HIV infections. The technology could also be used to combat other infectious diseases, such as hepatitis B and C, because the viruses share a similar protective membrane to the HIV virus.
The use of this technology in humans has yet to be explored and will require years of study and clinical trials to see if they are effective in real live people. The Healthline News team is committed to delivering content that adheres to the highest editorial standards for accuracy, sourcing, and objective analysis.
Every news article is thoroughly fact-checked by members of our Integrity Network. Furthermore, we have a zero-tolerance policy regarding any level of plagiarism or malicious intent from our writers and contributors. All Healthline News articles adhere to the following standards: All referenced studies and research papers must be from reputable and relevant peer-reviewed journals or academic associations.
All studies, quotes, and statistics used in a news article must link to or reference the original source. The article must also clearly indicate why any statistics presented are relevant.
All content related to new treatments, drugs, procedures, and so on must clearly describe availability, pricing, side effects, treatment target e. This immunotoxin targets HIV-infected cells and, when taken inside cells, shuts down protein synthesis and triggers cell death. For the new study, the NIH researchers teamed with Dr. Victor Garcia and colleagues at the University of North Carolina School of Medicine, where the experiments were performed.
The scientists studied 40 mice that were bioengineered to have a human immune system. The mice were infected with HIV. After several months, the mice were given a combination of antiretroviral drugs for 4 weeks. Half the animals subsequently received a 2-week dose of the 3B3-PE38 immunotoxin to complement the antiretrovirals, while the other half continued receiving antiretrovirals alone. Compared to antiretrovirals alone, the addition of the immunotoxin significantly reduced the number of cells with detectable virus in multiple organs.
It also lowered the level of HIV in the blood. These and previous findings suggest that immunotoxin treatment, when added to antiretroviral therapy, could help keep HIV in remission. The ultimate goal for such treatments would be to eliminate or control HIV infections well enough to allow people to live without a lifetime of continuous antiretroviral therapy.
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